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Endometrosis

Pathophysiology

Ten percent of women will develop endometrosis characterized by the presence of endometrial tissue at sites outside the endometrosis, pelvic pain, dysmenorrhea, menstrual pain uterine cavity. The ectopic endometrial cells cause the cyclical dysmenorrhea of endometrosis endometrosis endometreosis.

The most common sites are the ovaries, posterior cul-de-sac, uterosacral ligaments, posterior broad ligament, and anterior cul-de-sac. The uterine serosa, rectovaginal septum, cervix, vagina rectosigmoid, endometrosis, pelvic pain, dysmenorrhea, menstrual pain and bladder are less frequent locations.

Clinical Manifestations

endometrosis is characterized by cyclical pain, usually beginning prior to menses. Deep dyspareunia and sacral backache with menses are common.

Infertility is a frequent consequence of endometrosis. Premenstrual tenesmus or diarrhea may indicate rectosigmoid endometrosis. Cyclic dysuria or hematuria may indicate bladder endometrosis.

Diagnosis

Tender nodules are often palpable through the posterior vaginal fornix on bimanual examination and along the uterosacral ligaments on rectovaginal endometrosis endometrosis examination. Ovarian enlargement, endometrosis, pelvic pain, dysmenorrhea, menstrual pain endometrosis, pelvic pain, dysmenorrhea, menstrual pain fixation of the adnexal endometrosis, pelvic pain, retrodisplacement may also be detected.

Ultrasound may identify adnexal masses. Endometrosis can be definitively treated with